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The complementary effects of GLP-1 and GIP activation result in more comprehensive metabolic remodeling

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[1,2] More specifically, it's a GLP-1R agonist it activates the receptor, rather than inhibiting it

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The dissociated Epac2 then dimerizes with Rim2 and Piccolo in a Ca 2+ -dependent manner followed by induction of exocytosis of the insulin granules through binding of the Epac2/Rim2/Piccolo complex to Rab3 which is located at the membrane of the insulin granules (Figure 6) [508,511]