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The deficiency or mutation of the enzyme has been linked with various disease conditions and abnormalities.39 Zmock and Koller associated oxidative DNA damage and subsequent risk of cancer susceptibility in individuals with genetic polymorphisms and altered gene expression/activity in catalase.40 Development of mental disorder has also resulted from polymorphism in the catalase-encoding gene.41 In addition, humans with low catalase levels (acatalasemia) are said to be more predisposed to type 2 diabetes mellitus (Fig

The net result of these actions is inhibition of C-fibre-evoked postsynaptic excitation, although TRPA1-mediated calcium influx may initially increase spontaneous excitatory postsynaptic currents via activation of ionotropic glutamate receptors (iGluR)

The CO23strain was isolated from a subject with vulvovaginal candidiasis

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