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Description
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The brain is particularly vulnerable to oxidative damage, mainly because of its unique lipid composition, resulting in an amplified cascade of redox reactions that target several cellular components/functions ultimately leading to neurodegeneration
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At a histopathological level, reduced amyloid plaque deposition and microglial activation was seen, resulting in reduced neuroinflammation
Mitochondrial free radical overproduction due to respiratory chain impairment in the brain of a mouse model of Rett syndrome: protective effect of CNF1
doi: 10.1097/00024382-199608000-00002 66
