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In a monosodium iodoacetate-induced OA rat model, Methyltransferase-like 14 (METTL14) was confirmed to inhibit ferroptosis and ECM degradation in damaged chondrocytes by inhibiting m6A modification of GPX4 mRNA, a common RNA methylation, thereby protecting cartilage (Liu D

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The resulting mutant (all 6 residues were mutated in alanine) was correctly expressed and addressed to the membrane (Supplementary Fig

Alternative volumes for different resulting concentrations Rule: For AOD-9604 we recommend 2 ml as the optimal compromise volume