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Previous research has shown that excessive nitric oxide (NO) production, increased levels of oxidative stress, and cerebrovascular hypoperfusion are mayor pathomechanisms responsible for AD (Lepara et al

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Dang VT, Shi Y & Werstuck G (2015) Comprehensive metabolomic analysis of diabetic atherosclerosis

BPC-157 has demonstrated cytoprotective and wound-healing properties across gastrointestinal, musculoskeletal, and neurological models, acting through VEGF-mediated angiogenesis and nitric oxide system modulation

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