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Elevated levels of TMAO are linked to an increased risk of atherosclerosis (hardening of the arteries) and kidney disease

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Together, these findings indicate that AX enhances skeletal muscle recovery and function through PGC-1/TFAM-mediated mitochondrial biogenesis and activation of muscle-specific differentiation pathways [12]
The degree of cytologic atypia and architectural disorder (i.e., dysplasia) can be graded as mild, moderate, or severe and has been correlated with cell cycle marker expression (3)

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