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[DOI] [PMC free article] [PubMed] [Google Scholar] 144.McWilliams T.G., Prescott A.R., Montava-Garriga L., Ball G., Singh F., Barini E., Muqit M.M., Brooks S.P., Ganley I.G
Safety, pharmacokinetic, and pharmacodynamic evaluation of a 2-(2-methoxyethyl)-d-ribose antisense oligonucleotide-triantenarry n -acetyl-galactosamine conjugate that targets the human transmembrane protease serine 6
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fumigatus offered insight into the observed rise in EGT levels in the gliotoxin-deficient mutant gliK ( gliK was demonstrated to exhibit significant sensitivity to H 2 O 2 compared to wild-type (Figure 3) ( gliK deletion may result in a GSH sink, as GSH is committed to the defective gliotoxin biosynthetic pathway, resulting in compromised intracellular GSH content

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