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A major regulatory input to FOXO activity comes from the PI3K/Akt pathway and growth factor-dependent stress signaling [192]

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& Bruchovsky, N

Further development of TGR-based therapeutic approaches is impeded by the lack of non-covalent small molecule inhibitors of TGR, a well-known and general problem for all the members of the high molecular weight, Sec-containing TrxR subfamily that are considered attractive and unexploited drug targets for several human diseases 21,22