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Another interesting observation in this paper is that the gastric distension caused by GLP-1 activation of intestinofugal neurons is signaled via spinal and not vagal afferent pathways (398)
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It activates several overlapping pathways, notably VEGFR2 and nitric oxide synthesis via the Akt-eNOS axis, promoting angiogenesis, fibroblast activity, and neuromuscular stabilization
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