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Investigations show growing human use despite absent clinical data (Good Morning America, 2025

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Recent reports have shown that, in addition to platelet aggregation and activation, -toxin induces platelet apoptosis, platelet-neutrophil aggregate formation, aggregated platelet deposition in the liver, and initiates platelet protein synthesis (84, 8992)

Elevated levels of both metabolites are expected in vitamin B12 deficiency, whereas in folate deficiency, only increases in serum homocysteine concentrations occur