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These findings clearly show that BPC 157 may successfully compete with the initial events in intra-abdominal hypertension (i.e., significant damage to the intestinal epithelium and dilation of intestinal tight junctions, increased mucosal barrier permeability, bacterial translocation, and sepsis (Gong et al., 2009))

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The researchers of this study were based at the University of Bergen in Norway, conducting the study completely independent of any commercial interest

[DOI] [PubMed] [Google Scholar] 65.Wanrooij S., Fuste J.M., Farge G., Shi Y., Gustafsson C.M., Falkenberg M

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