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Further evidence supporting the potential in situ deleterious effects of APAP in brain comes from studies showing that APAP increases neuronal CYP2E1 enzymatic activity and protein levels, leading to neuronal cell death through mitochondrial-mediated mechanisms that involve cytochrome c release and caspase 3 activation (130)

Nanocarrierbased delivery systems (e.g., liposomes, polymeric nanoparticles, solid lipid nanoparticles) improve the solubility, bioavailability, and tumortargeting of phytochemicals, thereby enhancing their therapeutic index as adjuncts to conventional therapy (cited [184, 185, 186])