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These findings suggest that the activation of p58IPK appears to be a sequel to the Hsp70-mediated release of Hdj1 from the p58IPK-Hdj1 complex, which allows the monomeric p58IPK to inhibit PKR
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Nevertheless, current GSH-activated prodrugs frequently face challenges in achieving selective activation within heterogeneous tumor microenvironments
Dosage and frequency vary based on the patients condition and medical needs
Jun 2018;62:1-10
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