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Tendon tissue microdamage and the limits of intrinsic repair

Significant targets and the effects of PKGI in SMC are activation of myosin light chain phosphatase, inhibition of PLC, inhibition of the IP 3 receptor, inhibition of plasma membrane localized L-type Ca 2+ channels, and activation of plasma membrane localized calcium-activated K + channels (commonly called the big potassium, BK, channels)

[DOI] [PubMed] [Google Scholar] 115.Halliwell B

29,30 Mercury ingestion forms vacuoles in the anterior capsule that migrate posteriorly and become aggregated, affecting the sodium-potassium pump (Na+/K+-ATPase) and resulting in a rapidly increasing nuclear sclerotic cataract

doi: 10.1016/j.drudis.2007.08.010

The FOXO4 transcription factor, which usually helps cells repair themselves or undergo apoptosis when damaged, gets sequestered by p53 in senescent cells