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extrahepatic origin) is deeply affected by the clinical and epidemiological context

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DISCUSSION The intracellular ROS produced by paraquat was suppressed by extracellular GSH at concentrations of 1-10 mM (Fig

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SIRT1 further inhibits sterol regulatory element-binding protein 1c (SREBP-1c) to reduce fatty acid synthesis and activates peroxisome proliferator-activated receptor (PPAR) to promote fatty acid oxidation