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To further investigate the role of REST in AKI, both IRI- and cisplatin-induced AKI mouse models were established (Supplemental Figure 2, AF)
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Itaconic acid derivatives have been shown to enhance ferroptosis by stimulating autophagy, thereby diminishing the activity of caspases, key mediators of apoptosis, and reversing the efficacy of chemotherapeutic agents such as carboplatin, etoposide, and vincristine (220)
In vitro, endocrine and palbociclib resistant cells show increased OXPHOS dependency and increased ROS levels upon IACS-010759 treatment
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