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The beneficial microflora can also produce many metabolic products that moderate the interaction between the epithelium and the immune system and generate antimicrobial substances, inhibiting pathogens [14]
Given that both NCR-1 and NCR-2 function in neurons (Fig 5), we used a pan-neuronal promoter ( F25B3.3p ) to overexpress NCR-1::GFP and NCR-2::GFP specifically in neurons (Fig 6A)
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However, structures of LPLA2 bound to fluorophosphonate inhibitors are expected to mimic properties of the covalent intermediate of the reaction 23 (Table 1 and Supplementary Fig
