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Indeed, the most recognized effect of NO on skeletal muscle metabolism is its capacity to directly inhibit mitochondrial electron transport chain complexes: NO inhibits electron transfer and NADH-dehydrogenase function at the level of Complex I through the intra-mitochondrial production of ONOO [67]
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doi: 10.3892/ol_00000162
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Keywords: BPC 157, molecular pathways, efficacy and application, safety, BCP 157-based patents 1
Sympathetic nervous system overactivity and its role in the development of cardiovascular disease
