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Recently, we have demonstrated that dose- and time-dependent ASA treatment of HepG2 cells caused cell cycle arrest, increase in ROS production, reduction in mitochondrial potential, and alterations in mitochondrial redox and respiratory functions [23]

See Thioredoxin-related protein of 14 kDa is an efficient L-cystine reductase and S-denitrosylase, A novel persulfide detection method reveals protein persulfide- and polysulfide-reducing functions of thioredoxin and glutathione systems, and Thioredoxin-related protein of 14 kDa as a modulator of redox signalling pathways for further information

A further limitation is that our spectroscopic analysis was limited to the dorsal ACC and did not include more anterior/ventral portions of the medial prefrontal cortex

Natalizumab versus fingolimod for patients with active relapsing-remitting multiple sclerosis: results from REVEAL, a prospective, randomised head-to-head study

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