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There is either a reduced capacity of the mitochondria to buffer oxidation, or there is a persistent ROS production after treatment due to oxidative damage to the electron transport chain [169]

Subbian S, Tsenova L, Holloway J, Peixoto B, OBrien P, Dartois V, Khetani V, Zeldis JB, Kaplan G

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doi: 10.3390/molecules21101392 139

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[DOI] [PMC free article] [PubMed] [Google Scholar] 4.Armesto XL