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Our results partly support the hypothesis that accelerated cellular aging in individuals with psychiatric conditions could be a reversible process 66 , and that some of these effects, to the extent they exist, may be mediated via mechanisms involving telomerase and/or an antioxidant enzyme systems

Similarly, in a study of human subjects in New Zealand, selenium supplementation was found to increase GPx1 activity in patients with the Pro/Pro genotype compared to those with a Leu allele but only if they had a lower baseline plasma selenium concentration [38]

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