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[DOI] [PMC free article] [PubMed] [Google Scholar] 32.Pruteanu L.L., Bailey D.S., Grdinaru A.C., Jntschi L

Parasites Wildlife 20 GoutteA.AlliotF.BudzinskiH.Simonnet-LapradeC.SantosR.LachauxV.et al (2020)

doi: 10.1038/jid.2010.433

Diminished GSH synthesis caused by deficiency of its precursors cysteine and glycine is an essential contributor to GSH deficiency

This can, in theory, stave off diabetes

The activated Nrf2 promotes the transcription of antioxidant enzymes, including quinone oxidoreductase 1 (NQO1), heme oxygenase 1 (HO-1), and microsomal epoxide hydrolase, and further promotes GSH synthesis ( Once activated, the p53 tumor suppressor protein promotes cell survival and repairs genetic damage in response to various stress, such as cellular stress and DNA damage
