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Currently, mounting evidences suggest that there is a reciprocal influence between the gut and the liver, known as the gutliver axis [43], since the gut microbiota, microbial metabolites, and reciprocal interaction with the immune system influence the outcome of different liver disease
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SLC25A43 KO in HER2-positive breast cancer cells (BT-474) increases proliferation and cell cycle progression via enhanced G1-to-S transition, suggesting a role for SLC25A43 as a regulator of the cell cycle through a putative mitochondrial checkpoint [232]