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(Chichester) 2008;14(6):401409

Four alternative mechanisms might be used to explain how LC reduced TMX-induced brain injury: (1) LC directly removed ROS, suppressed brain cell lipid peroxidation, protected the cell membrane from oxidative stress, and maintained normal cell structure and functions

Similarly, nitrotyrosine/PECAM-1 ratios demonstrated a significant reduction with PBM treatment (ANOVA: F = 4.951, p = 0.0020, R 2 = 0.2921), highlighting its protective effects on endothelial cells under oxidative stress conditions (Fig

First, nearly all of the nLDs with a prominent coat of heterochromatin are near regions of the envelope that appear to be relatively deficient in peripheral heterochromatin (Fig 2C)

Although these mechanisms are part of normal healing, excessive or prolonged inflammation can cause serious complications (Jakob, 2001)

[DOI] [PubMed] [Google Scholar] Takaya K, Ogawa Y, Isse N, Okazaki T, Satoh N, Masuzaki H, Mori K, Tamura N, Hosoda K & Nakao K 1996