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6a), and the FBP-mediated conformational changes in RCC2 localized to a loop facing the presumed interface for Rac1 interaction (Fig

Demonstration that polyol accumulation is responsible for diabetic cataract by the use of transgenic mice expressing the aldose reductase gene in the lens

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Thereby, based on the obtained beneficial evidence, we claimed that with BPC 157 therapy, cytoprotection [26,27,28,29,30] as a particular vascular effect [26,27,28,29,30], wound healing [6,7,8,9,10], and neuroprotection [1,5,37] combine the principle of upgrading minor vessels [26,27]

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