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Description
Crucially, specific gut bacteria directly influence thyroid hormone metabolism through intrinsic enzymatic activities, such as bacterial deiodinase-like enzymes
HCV core protein interacts with the mitochondrial chaperone protein prohibitin, increasing its stability and levels but impairing its ability to interact with cytochrome c oxidase (COX) subunits, potentially resulting in disrupted COX assembly that could lead to increased ROS [101]

As discussed by Cakir et al

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Additionally, the intervention affected the cell cycle progression and apoptosis, and alleviated metabolic disorders (Yu et al., 2020)

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