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In a similar study, the pivotal role for the IGF-1 in insulin sensitivity has received further support from liver-specific IGF-1 KO mice which exhibited overt insulin resistance and hyperinsulinaemia that was reversed by the administration of IGF-1 [161]

1g), but only significantly downregulated the hypoxia-induced increase in secretion of d-serine, not l-serine (Fig

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