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Thioacetamide-induced renal oxidative stress decreases mitochondrial biogenesis, and increases autophagy and inflammatory, apoptotic, and fibrotic responses [128]

doi: 10.1007/s10072-009-0074-2

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Patients with kidney disease, congestive heart failure, or genetic conditions such as G6PD deficiency face higher risks and should avoid elective infusions unless prescribed
