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When co-administered with methyl donors and carnitine, it provides the acetyl acceptor capacity necessary for sustained mitochondrial oxidation, creating a complementary, multi-pathway approach to lipid mobilization and energy production

doi: 10.1016/0024-3205(82)90677-4

Strategies to optimize mitochondrial function could play an important role in limiting disease initiation and progress

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This was supported by the evidence that the genetic removal of Mfn2 in POMC neurons results in severe obesity characterized by excessive eating and reduced energy expenditure (66)