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Importantly, overexpression of keap1a or knockdown of hmox1a partially rescued the blood clot formation, suggesting that the disrupted Keap1-Nrf2 signaling is responsible for the venous thrombosis-like phenotypes in smarca5 mutants

BPC-157 & TB-500 Research Peptide SET:This set contains research-grade peptides BPC-157 and TB-500 intended solely for laboratory and scientific research purposes