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Upon re-entry into mitosis, the Aurora AHDAC6 (Histone deacetylase 6) pathway triggers ciliary disassembly through tubulin deacetylation [96]

Steinkamp, P

At the onset of RA, a large number of inflammatory factors such as TNF-, IL-6, IL-17, and IL-1 accumulate in the joints, causing inflammatory storms (121)

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Curr Neurovasc Res 10(4):415424 Dugger BN, Dickson DW (2017) Pathology of neurodegenerative diseases

-L-glutamyl-L-cysteine can be a substrate for -glutamylcyclotransferase (GCT), however, due to higher affinity and activity of glutathione synthetase (GS), glutathione synthesis is favored in animal cells.[118] In the presence of GS and ATP, -L-glutamyl-L-cysteine is phosphorylated to form enzyme bound -glutamyl-cysteinyl-(P), which reacts with Gly to liberate free glutathione and Pi.[9] This pathway occurs virtually in all cell types (except epithelial cells), with liver being the major producer and exporter of glutathione