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J Neurosci 17(8):26532657

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WEHI, 32D (32Dcl3), WT, T315I-BcrAbl-transformed 32D, and TCCS cells were maintained in RPMI 1640 medium (Sigma) supplemented with 10% heat-inactivated FBS, 1% Streptomycin/Penicillin, 1 mM sodium pyruvate, and 10 mM HEPES

They found that miR-200 targeted FOG2 which binds to p85 and negatively regulate PI3K activity suggesting that miR-200 regulates cell proliferation by regulating PI3K-AKT signaling activity

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