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[DOI] [PubMed] [Google Scholar] 32.Dyson HJ, Jeng MF, Tennant LL, Slaby I, Lindell M, Cui DS, Kuprin S, Holmgren A

As part of its wound healing effect, GHK-Cu has been shown to contribute to the initial upregulation of growth factors and inflammatory mediators during nerve regeneration [8]

Neuronal GHS-R deletion simultaneously promotes both spontaneous and voluntary activity, and has differential effects in different dopaminergic neurons in midbrain 9

QU can inhibit the activity of MMP-2, thereby inhibiting the degradation of the basement membrane and indirectly inhibiting angiogenesis and cartilage damage (Haleagrahara et al

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Among identified risk factors, exposure to pesticides and traumatic brain injury have received substantial support in the scientific literature (11)