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Taken together, c-Met could be targeted using selective and non-selective inhibitors, monoclonal or bispecific antibodies, and miRNAs to inhibit tumor progression

The goal here is no longer rapid replenishment but steady upkeep

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doi: 10.2147/CCID.S374295
However, they have low or absent expression levels of drug-metabolizing enzymes, impaired host defense system, and loss of the NTCP receptor (Figure 5)

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