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Apart from these shared features, other genetic alterations, such as the loss of PTEN , NOTCH receptors and the CREB binding protein, are also observed and the importance of these defects has been functionally validated in vivo and in vitro [126]

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The headline mechanisms: Angiogenesis via VEGFR2/Akt/eNOS: This is clinically significant because tendons and ligaments are notoriously hypovascular