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In vivo studies using mice lacking a component of the base excision repair process revealed significant stomach lesions after H pylori infection ( H

Reconstitution of FSP1 expression recovered the anti-ferroptotic function of phylloquinone and MK4, whereas expression of the myristoylation-defective G2A mutant of FSP1 4 did not rescue the protective effects of phylloquinone and MK4 in FSP1-KO cells (Fig

These products may affect cell DNA and increase the expression of genes that promote RPE cell aging [22]