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Liver 8: 151156, 1988 [DOI] [PubMed] [Google Scholar] 57.Mannery YO, Ziegler TR, Park Y, Jones DP: Oxidation of plasma cysteine/cystine and GSH/GSSG redox potentials by acetaminophen and sulfur amino acid insufficiency in humans

In summary, targeted silencing of CBS substantially suppressed the proliferation of STAD cells, diminished GSH production, and provoked oxidative stress within the STAD cells

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While it is acknowledged that ferroptosis is initiated by the peroxidation of PUFAs in the cellular membrane and organellar membranes such as the endoplasmic reticulum, the precise mechanisms through which these processes lead to cell death remain uncertain

2 The most studied example of Type I- GS is from studies of Bacillus subtilis GS which displays a strict product inhibition mechanism

Activation of the NRF2 antioxidant program sensitizes tumors to G6PD inhibition