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Additionally, the histological changes, sperm count, and miRNA expression (miR-155, miR-155-5p, and miR-132) were also studied

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Mechanistic investigation revealed ChaC1/AUR-driven cytotoxicity was attenuated by disulfide reducing agents (e.g., NAC and TCEP), while maintaining resistance to canonical programmed death pathway inhibitors targeting apoptosis, necroptosis, and ferroptosis
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