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[DOI] [PubMed] [Google Scholar] 105.Newell SM, Selcer BA, Girard E

Mutants E419A and R571A could process completely, while H401A and T417A showed 6070% processing in a time-dependent fashion, and T415A mutant persisted as an inactive precursor

PM2.5 contributed to pulmonary epithelial senescence and ferroptosis by regulating USP3-SIRT3-P53 axis

Meanwhile, ATP detection assays confirmed that the ATP level in GGCT-knockdown cells was significantly reduced and rescued in sodium pyruvate or JX06 treatment groups (Fig

Redox proteomics of protein-bound methionine oxidation

Peroxiredoxin-2 expression is increased in beta-thalassemic mouse red cells but is displaced from the membrane as a marker of oxidative stress