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Results Despite impaired cardiolipin remodelling, pancreatic islets from Taz-KD mice maintained insulin secretion, supported by compensatory mechanisms such as increased glucose uptake, expanded mitochondrial volume and increased metabolic parameters

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Chronic administration of AOD9604 in obese mice and rats leads to significant reductions in body weight gain and increases in fat oxidation, without the adverse effects on insulin sensitivity seen with full-length hGH treatment [3][4]

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