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Possibly, with respect to the underlying disease (which was markedly counteracted in BPC 157 rats), more activity of eNOS (the constitutive isoforms instantly increase along with COX2), and more activity of nNOS (NO produced by nNOS is thought to mediate synaptic plasticity and neuronal signaling) [103,104], may be intrinsic effects of BPC 157

The study was conducted and reported following the recommendations of the Consolidated Standards of Reporting Trials (CONSORT) statement

Members of the super family have long been known to be overexpressed in many types of cancers [95,96,97]

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