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Within controlled research environments, measurable outcomes such as altered GH pulse amplitude, downstream IGF-1 pathway modulation, and changes in cellular repair signaling have been documented without the disruptive hormonal cascades seen in less selective secretagogues

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Here, we profile key ferroptotic defence strategies including iron regulation, phospholipid modulation and enzymes and metabolite systems: glutathione reductase (GR), Ferroptosis suppressor protein 1 (FSP1), NAD(P)H Quinone Dehydrogenase 1 (NQO1), Dihydrofolate reductase (DHFR), retinal reductases and retinal dehydrogenases (RDH) and thioredoxin reductases (TR)