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Case of adult-onset type II citrullinemia treated as schizophrenia for a long time

Finally, to further demonstrate that unc1 is acting neuronally, we disrupted unc1 function across the nervous system by overexpressing a cDNA containing the n494 dominant allele of unc1 , again using the rab3 promoter to drive expression in all neurons

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Preformulation studies with melanotan-II: a potential skin cancer chemopreventive peptide

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Eskandari, Cancer exosomes and natural killer cells dysfunction: biological roles, clinical significance and implications for immunotherapy