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The mechanism cited most often in the preclinical literature is upregulation of vascular endothelial growth factor signaling (VEGFR2) alongside modulation of the nitric-oxide system

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It is under investigation in preclinical and clinical studies on overweight, obesity, and metabolic disorders, where researchers have examined its effects on body weight and energy intake, including when combined with GLP 1 receptor agonists such as semaglutide, but it is not an approved therapy

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