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Continuously activated microglia may cause persistent inflammation, leading to neuronal damage and promoting the formation of amyloid beta (A) plaques [43, 44]

Biochim Biophys Acta 1833(6):14981510

We have shown here using the unique advantages of the zebrafish embryo/larval model for in vivo imaging and drug/genetic screening a crucial contribution of NAMPT and PARP1 to skin inflammation through the induction of parthanatos cell death (Fig 8)

Additionally, endothelium-derived NO inhibits platelet adhesion, leukocyte infiltration, and oxidative stress, thereby contributing to vascular homeostasis 8,9

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Employing digital PCR (dPCR) and next-generation sequencing to longitudinally quantify mtDNA mutations in paired peripheral blood, tissue biopsies, and CSF will clarify their roles in residual inflammation, neurocognitive decline, and immunological failure