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Description
Reprogramming of mitochondrial serine metabolism in colorectal cancer facilitates purine nucleotide biosynthesis and enables drug-resistant cells to prevent the accumulation of drug-induced DNA damage, thereby promoting resistance to 5-FU (54)
Conclusions As molecular subtyping becomes standard in EC, it is important to understand the limitations of traditional IHC and methylation testing as well as the heterogeneity within the MMR-D/MSI-H group

Caniglia et al

Want to maximize recovery within 24 hours of a hard training block

Nutr Cancer 1994

The organisms body has developed a different endogenous system to counteract the generation of reactive oxygen intermediates
