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Increased iron levels and accumulation of lipid peroxidation products have been observed in the hippocampus and cortex of AD patients

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Co-cultures of glioma and neuronal cells demonstrated that the released l-Glu was sufficient to induce excitotoxic damage and that the neuronal loss could be reduced by either by preventing its efflux through Sx c - (4- S -CPG or SSZ) or inhibiting its action at NMDA receptors (MK-801)

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