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These findings suggest that adequate exportation of Gln from astrocytes represents a crucial regulatory point for maintaining GABA levels in the brain, achievable solely via the function of both SNAT-3 and SNAT-5

Hyperphosphorylation of several TDP-43 CTD serine residues via casein kinase 1 was previously reported occur at disease-associated sites, likely promoting TDP-43 aggregation 89

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(4) GSH aided in the increased rate of photosynthesis

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Advanced glycation end products (AGEs) The content of AGEs was determined through spectrofluorometry