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In a model of methotrexate-induced kidney injury, the administration of UMB reduced the expression of inflammation mediators P 38 MAPK and NF-B and the pro-apoptotic molecules BAS and caspase3 while increasing anti-apoptotic levels of the BLC-2 molecule [86]
The treated animals demonstrated better collagen fiber alignment, increased tensile strength, and faster return to normal biomechanical function
ISBN 978-94-011-4439-1
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