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PLA2G16-mediated tetracosatetraenoic acid rewires fatty acid oxidation to impair CD8 + T cell immune function in promoting breast cancer lung metastasis

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Rather than merely fueling tumor growth, this extensive metabolic rewiring profoundly reshapes the tumor microenvironment (TME), establishing complex metabolic crosstalk that actively drives immune evasion

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Moreover, the association of virions with lipoproteins and the presence of a glycan shield (possessed by the E1-E2 heterodimers), reduce the antibody access to the sensitive neutralizing epitopes[24,41,42]